MiR-690 treatment causes decreased fibrosis and steatosis and restores specific Kupffer cell functions in NASH
Hong Gao(Zhengzhou University), Jerrold M. Olefsky(University of California, San Diego), David A. Brenner(University of North Carolina at Chapel Hill), Tatiana Kisseleva(University of California San Diego), Hani Jouihan(Johnson & Johnson (United States)), Dinghong Zhang(Chinese Academy of Sciences), Karina Cunha e Rocha(University of California San Diego), Soheil Pourshahian(Johnson & Johnson (United States)), Wei Ying(Texas A&M University), Zhongmou Jin(University of California, San Diego), Huayi Zhao(University of California, San Diego), Xiao Liu(University of California, San Diego), Gautam Bandyopadhyay(University of California San Diego)
Cited by 123
Related Papers
Liver fibrosis
|Journal of Clinical Investigation|2005|4.7k
Insulin sensitivity: modulation by nutrients and inflammation
|Journal of Clinical Investigation|2008|1.2k
A Subpopulation of Macrophages Infiltrates Hypertrophic Adipose Tissue and Is Activated by Free Fatty Acids via Toll-like Receptors 2 and 4 and JNK-dependent Pathways
|Journal of Biological Chemistry|2007|1k
Exosomes as mediators of intercellular crosstalk in metabolism
|Cell Metabolism|2021|810