Defective <scp>fractalkine‐CX3CR1</scp> signaling aggravates neuroinflammation and affects recovery from cuprizone‐induced demyelination
Andrew S. Mendiola(Gladstone Institutes), Astrid E. Cardona(The University of Texas at San Antonio), Kaira A. Church(Texas Center for Infectious Disease), Richard M. Ransohoff(Cleveland Clinic Lerner College of Medicine), Sérgio A. Lira(Tisch Hospital), Chin‐Hsing Annie Lin(The University of Texas at San Antonio), Erzsebet Kokovay(The University of Texas at San Antonio Health Science Center), Shannon A. Garcia(The University of Texas at San Antonio), Sandra M. Cardona(The University of Texas at San Antonio), Wendy B. Macklin(University of Colorado Anschutz Medical Campus), Difernando Vanegas(Texas Center for Infectious Disease)
Cited by 17
Related Papers
Constitutively Active Akt Induces Enhanced Myelination in the CNS
|Journal of Neuroscience|2008|361
Akt Signals through the Mammalian Target of Rapamycin Pathway to Regulate CNS Myelination
|Journal of Neuroscience|2009|321
The IL-1β phenomena in neuroinflammatory diseases
|Journal of Neural Transmission|2017|301