Insulin-like Growth Factors Mediate Heterotrimeric G Protein-dependent ERK1/2 Activation by Transactivating Sphingosine 1-Phosphate Receptors
Hesham M. El‐Shewy(Medical University of South Carolina), Louis M. Luttrell(Medical University of South Carolina)
Cited by 26
Related Papers
Role of c-Src Tyrosine Kinase in G Protein-coupled Receptorand Gβγ Subunit-mediated Activation of Mitogen-activated Protein Kinases
|Journal of Biological Chemistry|1996|531
Beyond Desensitization: Physiological Relevance of Arrestin-Dependent Signaling
|Pharmacological Reviews|2010|362
The β-Arrestin Pathway-selective Type 1A Angiotensin Receptor (AT1A) Agonist [Sar1,Ile4,Ile8]Angiotensin II Regulates a Robust G Protein-independent Signaling Network
|Journal of Biological Chemistry|2011|70
Role of β-Arrestin-mediated Desensitization and Signaling in the Control of Angiotensin AT1a Receptor-stimulated Transcription
|Journal of Biological Chemistry|2007|57
β-Arrestin 2 Expression Determines the Transcriptional Response to Lysophosphatidic Acid Stimulation in Murine Embryo Fibroblasts
|Journal of Biological Chemistry|2005|46