Inhibition of endogenous thioredoxin in the heart increases oxidative stress and cardiac hypertrophy
Mitsutaka Yamamoto(Rutgers New Jersey Medical School), Junichi Sadoshima(Rutgers, The State University of New Jersey)
Cited by 235
Related Papers
Autophagy in major human diseases
|The EMBO Journal|2021|1.6k
Silent Information Regulator 2α, a Longevity Factor and Class III Histone Deacetylase, Is an Essential Endogenous Apoptosis Inhibitor in Cardiac Myocytes
|Circulation Research|2004|302
Nicotinamide Mononucleotide, an Intermediate of NAD+ Synthesis, Protects the Heart from Ischemia and Reperfusion
|PLoS ONE|2014|295
Activation of Mst1 causes dilated cardiomyopathy by stimulating apoptosis without compensatory ventricular myocyte hypertrophy
|Journal of Clinical Investigation|2003|277
A functional interaction between Hippo-YAP signalling and FoxO1 mediates the oxidative stress response
|Nature Communications|2014|274