Disuse-associated loss of the protease LONP1 in muscle impairs mitochondrial function and causes reduced skeletal muscle mass and strength
Zhisheng Xu(Nanjing Drum Tower Hospital), Zhenji Gan(Sanford Burnham Prebys Medical Discovery Institute)
Cited by 78
Related Papers
The nuclear receptor PPARβ/δ programs muscle glucose metabolism in cooperation with AMPK and MEF2
|Genes & Development|2011|135
Exercise Inducible Lactate Dehydrogenase B Regulates Mitochondrial Function in Skeletal Muscle
|Journal of Biological Chemistry|2016|103
Enhancer regulator MLL4 controls skeletal muscle metabolic efficiency by limiting AMPK-mediated fuel catabolism
|Nature Communications|2025|0