IκB-ζ signaling promotes chondrocyte inflammatory phenotype, senescence, and erosive joint pathology
Manoj Arra(Washington University in St. Louis), Yousef Abu‐Amer(Washington University in St. Louis)
Cited by 64
Related Papers
Aseptic loosening of total joint replacements: mechanisms underlying osteolysis and potential therapies
|Arthritis Research & Therapy|2007|526
Tumor Necrosis Factor-α (TNF) Stimulates RANKL-induced Osteoclastogenesis via Coupling of TNF Type 1 Receptor and RANK Signaling Pathways
|Journal of Biological Chemistry|2001|499
LDHA-mediated ROS generation in chondrocytes is a potential therapeutic target for osteoarthritis
|Nature Communications|2020|386
IL-4 abrogates osteoclastogenesis through STAT6-dependent inhibition of NF-κB
|Journal of Clinical Investigation|2001|188
IKKβ activation is sufficient for RANK-independent osteoclast differentiation and osteolysis
|Journal of Bone and Mineral Research|2010|55