Identification of an exporter that regulates vitamin C supply from blood to the brain
Hiroshi Miyata(Osaka University), Hiroshi Suzuki(University of Tokyo Hospital), Ryuichiro Shigesawa(University of Tokyo Hospital), Tappei Takada(University of Tokyo Hospital), Yuji Ikegaya(The University of Tokyo), Toshimitsu Hiragi(The University of Tokyo), Ryuta Koyama(The University of Tokyo), Yu Kubota(University of Tokyo Hospital), Yu Toyoda(University of Tokyo Hospital)
Cited by 19
Related Papers
Decreased extra-renal urate excretion is a common cause of hyperuricemia
|Nature Communications|2012|703
Common Defects of ABCG2, a High-Capacity Urate Exporter, Cause Gout: A Function-Based Genetic Analysis in a Japanese Population
|Science Translational Medicine|2009|438
Chk2‐deficient mice exhibit radioresistance and defective p53‐mediated transcription
|The EMBO Journal|2002|436
Genome-wide association study of clinically defined gout identifies multiple risk loci and its association with clinical subtypes
|Annals of the Rheumatic Diseases|2015|185