SARS‐CoV‐2 N Protein Induces Acute Kidney Injury via Smad3‐Dependent G1 Cell Cycle Arrest Mechanism
Wenbiao Wang(Guangdong Academy of Medical Sciences), Hui Y. Lan(Chinese University of Hong Kong, Shenzhen)
Cited by 86
Related Papers
TGF-β/Smad signaling in renal fibrosis
|Frontiers in Physiology|2015|648
miR-21 is a key therapeutic target for renal injury in a mouse model of type 2 diabetes
|Diabetologia|2013|353
The Protective Role of Smad7 in Diabetic Kidney Disease: Mechanism and Therapeutic Potential
|Diabetes|2010|224
Zika virus infection induces host inflammatory responses by facilitating NLRP3 inflammasome assembly and interleukin-1β secretion
|Nature Communications|2018|215
EV71 3D Protein Binds with NLRP3 and Enhances the Assembly of Inflammasome Complex
|PLoS Pathogens|2017|115