Enhanced glycolysis and GSK3 inactivation promote brain metabolic adaptations following neuronal mitochondrial stress
Sofía García(University of Miami), Francisca Díaz(University of Miami), Ami P. Raval(University of Miami), Nadee Nissanka(University of Miami), Amy Saldana-Caboverde(University of Miami), Carlos T. Moraes(University of Miami), Mir R. Anwar(University of Miami), Milena Pinto(University of Miami)
Cited by 15
Related Papers
MELAS: Clinical features, biochemistry, and molecular genetics
|Annals of Neurology|1992|508
Mitochondrial <scp>DNA</scp> damage and reactive oxygen species in neurodegenerative disease
|FEBS Letters|2017|441
Mitochondrial myopathy induces a starvation-like response
|Human Molecular Genetics|2010|306
MitoTALEN reduces mutant mtDNA load and restores tRNAAla levels in a mouse model of heteroplasmic mtDNA mutation
|Nature Medicine|2018|285
Atypical clinical presentations associated with the MELAS mutation at position 3243 of human mitochondrial DNA
|Neuromuscular Disorders|1993|238