Loss of the H4 lysine methyltransferase KMT5B drives tumorigenic phenotypes by depleting H3K27me3 at loci otherwise retained in H3K27M mutant DIPG cells
Ketty Kessler(Institute of Cancer Research), Chris Jones(Institute of Cancer Research)
Cited by 0
Related Papers
MGMT-Independent Temozolomide Resistance in Pediatric Glioblastoma Cells Associated with a PI3-Kinase–Mediated <i>HOX</i>/Stem Cell Gene Signature
|Cancer Research|2010|158
ALK2 inhibitors display beneficial effects in preclinical models of ACVR1 mutant diffuse intrinsic pontine glioma
|Communications Biology|2019|120
c-KIT overexpression, without gene amplification and mutation, in paediatric renal tumours
|Journal of Clinical Pathology|2007|29