A third-generation mouse model of Alzheimer's disease shows early and increased cored plaque pathology composed of wild-type human amyloid β peptide
Kaori Sato(Gunma University), Hiroki Sasaguri(RIKEN Center for Brain Science), Takaomi C. Saido(RIKEN Center for Brain Science), Toshio Ohshima(Waseda University), Naoto Watamura(UK Dementia Research Institute), Ryo Fujioka(RIKEN Center for Brain Science), Kenichi Nagata(Nagoya University), Misaki Sekiguchi(RIKEN Center for Brain Science), Naomi Mihira(RIKEN Center for Brain Science), Takashi Saito(Nagoya City University)
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