Post-Treatment with Amorfrutin B Evokes PPARγ-Mediated Neuroprotection against Hypoxia and Ischemia

Agnieszka Wnuk(Maj Institute of Pharmacology), Małgorzata Kajta(Maj Institute of Pharmacology)

Biomedicines

July 21, 2021

10.3390/biomedicines9080854

Cited by 12

Related Papers

Prenatal Exposure to Benzophenone-3 Impairs Autophagy, Disrupts RXRs/PPARγ Signaling, and Alters Epigenetic and Post-Translational Statuses in Brain Neurons

|Molecular Neurobiology|2018|62

Posttreatment with PaPE-1 Protects from Aβ-Induced Neurodegeneration Through Inhibiting the Expression of Alzheimer’s Disease-Related Genes and Apoptosis Process That Involves Enhanced DNA Methylation of Specific Genes

|Molecular Neurobiology|2023|4

Multigenerational Consequences of Prenatal Exposure to Benzophenone-3 Demonstrate Sex- and Region-Dependent Neurotoxic and Pro-Apoptotic Effects in Mouse Brain

|Toxics|2024|2