Prolyl-4-hydroxylase 3 maintains β cell glucose metabolism during fatty acid excess in mice
Daniela Nasteska(Oxford Centre for Diabetes, Endocrinology and Metabolism), David J. Hodson(University of Birmingham)
Cited by 8
Related Papers
Beta Cell Hubs Dictate Pancreatic Islet Responses to Glucose
|Cell Metabolism|2016|472
Revisiting the Complexity of GLP-1 Action from Sites of Synthesis to Receptor Activation
|Endocrine Reviews|2020|268
Super-resolution microscopy compatible fluorescent probes reveal endogenous glucagon-like peptide-1 receptor distribution and dynamics
|Nature Communications|2020|143
The role of beta cell heterogeneity in islet function and insulin release
|Journal of Molecular Endocrinology|2018|61