Coding and noncoding variants in EBF3 are involved in HADDS and simplex autism

Evin M. Padhi(Washington University in St. Louis), Tychele N. Turner(Washington University in St. Louis), Kévin Uguen(Université de Pau et des Pays de l'Adour), Sylvia Redon(Inserm), Shazia Maqbool, Riana D. Hunter(Lawrence Berkeley National Laboratory), Diane E. Dickel(Lawrence Berkeley National Laboratory), Jeffrey K. Ng(Washington University in St. Louis), Andrew S. Allen(Duke University), Fatima Rahman, Stéphanie Efthymiou(Queen Mary University of London), Séverine Audebert‐Bellanger(Centre Hospitalier Régional Universitaire de Brest), Tristan J. Hayeck(Broad Institute), David U. Gorkin(Emory University), L Pennacchio(Lawrence Berkeley National Laboratory), Sumantra Chatterjee(Center for Human Genetics), Michael C. Zody(Whitehead Institute for Biomedical Research), Marta Byrska-Bishop(New York Genome Center), Rajeeva Musunuri(New York Genome Center), Marjolaine Willems(Université de Montpellier), Brandon J. Mannion(Lawrence Berkeley National Laboratory), Raphael Bernier(University of Washington), Elvisa Mehinovic(Washington University in St. Louis), Lauren E. Fries(Columbia University), Claude Férec(Université de Bretagne Occidentale), Jennifer A. Akiyama(Lawrence Berkeley National Laboratory), Cédric Le Maréchal(Inserm), Giuseppe Narzisi(Cold Spring Harbor Laboratory), Henry Houlden(Queen Mary University of London), Reza Maroofian(University College London), Caroline Bénech(Inserm), Lucile Pinson(Université de Montpellier), Avinash Abhyankar(Rockefeller University)
Human Genomics
July 13, 2021
Cited by 35


Related Papers