Synaptic disruption and CREB‐regulated transcription are restored by K+ channel blockers in ALS
Alberto Catanese(Universität Ulm), Tobias M. Böckers(Universität Ulm)
Cited by 56
Related Papers
Shank2 identifies a subset of glycinergic neurons involved in altered nociception in an autism model
|Molecular Autism|2023|37
Shank2/3 double knockout-based screening of cortical subregions links the retrosplenial area to the loss of social memory in autism spectrum disorders
|Molecular Psychiatry|2022|20
Integrative proteomics highlight presynaptic alterations and c-Jun misactivation as convergent pathomechanisms in ALS
|Acta Neuropathologica|2023|18
Integrative proteomics highlight presynaptic alterations and c-Jun misactivation as convergent pathomechanisms in ALS
|Research Square|2023|1
Shank2 expression identifies a subpopulation of glycinergic interneurons involved in nociception and altered in an autism mouse model
|bioRxiv (Cold Spring Harbor Laboratory)|2020|0