Cancer-Associated Fibroblasts in Pancreatic Ductal Adenocarcinoma Determine Response to SLC7A11 Inhibition
George Sharbeen(Cancer Institute of New South Wales), Phoebe A. Phillips(UNSW Sydney)
Cited by 129
Related Papers
Ectopic restriction of DNA repair reveals that UNG2 excises AID-induced uracils predominantly or exclusively during G1 phase
|The Journal of Experimental Medicine|2012|55
Cancer-associated fibroblasts in pancreatic ductal adenocarcinoma determine response to SLC7A11 inhibition
|bioRxiv (Cold Spring Harbor Laboratory)|2020|9
βIII-Tubulin is a Brake on Extrinsic Cell-Death in Pancreatic Cancer
|bioRxiv (Cold Spring Harbor Laboratory)|2022|1
MYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability
|Neoplasia|2025|0
βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer
|Cell Death and Disease|2026|0