B7-H3 suppresses doxorubicin-induced senescence-like growth arrest in colorectal cancer through the AKT/TM4SF1/SIRT1 pathway
Ruoqin Wang(Soochow University), Weichang Chen(Chinese Academy of Sciences), Xueguang Zhang(Harbin Institute of Technology), Tongguo Shi(Soochow University), Suhua Xia(Soochow University), Guangbo Zhang(Soochow University), Linqing Sun(University of Pittsburgh), Shenghua Zhan(Soochow University), Hongya Wu(Ministry of Agriculture and Rural Affairs), Yanchao Ma(Soochow University)
Cited by 39
Related Papers
B7-H3 promotes aerobic glycolysis and chemoresistance in colorectal cancer cells by regulating HK2
|Cell Death and Disease|2019|243
B7-H3 promotes colorectal cancer angiogenesis through activating the NF-κB pathway to induce VEGFA expression
|Cell Death and Disease|2020|230
G-quadruplex dynamics contribute to regulation of mitochondrial gene expression
|Scientific Reports|2019|112
H19 promotes aerobic glycolysis, proliferation, and immune escape of gastric cancer cells through the microRNA‐519d‐3p/lactate dehydrogenase A axis
|Cancer Science|2021|82
B7-H3 inhibits the IFN-γ-dependent cytotoxicity of Vγ9Vδ2 T cells against colon cancer cells
|OncoImmunology|2020|81