Sensitivity of cells to ATR and CHK1 inhibitors requires hyperactivation of CDK2 rather than endogenous replication stress or ATM dysfunctionJennifer P. Ditano(Dartmouth College), Alan Eastman(Dartmouth College)Scientific ReportsMarch 29, 202110.1038/s41598-021-86490-xCited by 14SaveCiteExport RISWatch citationsRelated PapersA subset of cancer cell lines is acutely sensitive to the Chk1 inhibitor MK-8776 as monotherapy due to CDK2 activation in S phase|Oncotarget|2015|74