Melanoma subpopulations that rapidly escape MAPK pathway inhibition incur DNA damage and rely on stress signalling
Chen Yang(University of Colorado Boulder), Sabrina L. Spencer(University of Colorado Boulder)
Cited by 93
Related Papers
Multiple cancers escape from multiple MAPK pathway inhibitors and use DNA replication stress signaling to tolerate aberrant cell cycles
|Science Signaling|2023|23
Rapidly induced drug adaptation mediates escape from BRAF inhibition in single melanoma cells
|bioRxiv (Cold Spring Harbor Laboratory)|2020|7
BRAF <sup>V600</sup> and ErbB inhibitors directly activate GCN2 in an off-target manner to limit cancer cell proliferation
|bioRxiv (Cold Spring Harbor Laboratory)|2024|2
p16 expression confers sensitivity to CDK2 inhibitors in cyclin E1–driven ovarian cancers
|Science Signaling|2025|2
Multiple cancer types rapidly escape from multiple MAPK inhibitors to generate mutagenesis-prone subpopulations
|bioRxiv (Cold Spring Harbor Laboratory)|2023|1