Recruitment of γδ T cells to the lesion via the CCL2/CCR2 signaling after spinal cord injury
Ping Xu(First Affiliated Hospital of Jinan University), Guodong Sun(Zhejiang Entry-Exit Inspection and Quarantine Bureau), Jingchun Yao, Haoran Zhu(Army Medical University), Si-Tao Li(Sun Yat-sen University), Feng Zhang(First Affiliated Hospital of Jinan University), Zhizhong Li(Jinan University), Wen-Cai Zhang(First Affiliated Hospital of Jinan University), Cheng Zhong, Minmin Chang(Shandong University of Traditional Chinese Medicine), Chenghong Sun(Luye Pharma (China))
Cited by 52
Related Papers
hucMSC derived exosomes promote functional recovery in spinal cord injury mice via attenuating inflammation
|Materials Science and Engineering C|2018|351
ALKBH5-HOXA10 loop-mediated JAK2 m6A demethylation and cisplatin resistance in epithelial ovarian cancer
|Journal of Experimental & Clinical Cancer Research|2021|206
γδ T cells provide the early source of IFN-γ to aggravate lesions in spinal cord injury
|The Journal of Experimental Medicine|2017|133
Articular cartilage repair biomaterials: strategies and applications
|Materials Today Bio|2024|132
GLS1-mediated glutaminolysis unbridled by MALT1 protease promotes psoriasis pathogenesis
|Journal of Clinical Investigation|2020|118