SMN Depleted Mice Offer a Robust and Rapid Onset Model of Nonalcoholic Fatty Liver Disease
Marc‐Olivier Deguise(University of Ottawa), Rashmi Kothary(University of Ottawa)
Cited by 38
Related Papers
Myogenic program dysregulation is contributory to disease pathogenesis in spinal muscular atrophy
|Human Molecular Genetics|2014|83
A novel whole-cell lysate kinase assay identifies substrates of the p38 MAPK in differentiating myoblasts
|Skeletal Muscle|2012|52
Integrin-Linked Kinase Regulates Process Extension in Oligodendrocytes via Control of Actin Cytoskeletal Dynamics
|Journal of Neuroscience|2013|34
A mouse model for spinal muscular atrophy provides insights into non-alcoholic fatty liver disease pathogenesis
|bioRxiv (Cold Spring Harbor Laboratory)|2020|3