Mitochondria Form Contact Sites with the Nucleus to Couple Pro-Survival Retrograde Response
Radha Desai(University of London), Michelangelo Campanella(Centre National de la Recherche Scientifique), Manuel Rigon(William Harvey Research Institute), Valentina Zappulli(University of Padua), María Soledad Álvarez(University of London), Lisa E. L. Romano(Queen Mary University of London), Robert B. Bentham(MRC Laboratory for Molecular Cell Biology), Liana Hardy(University of London), James Crosby(University of London), Danilo Faccenda(University of London), Xia Dong(University of London), Ken Smith(University of Utah), Marta Mainenti(Iowa State University), Gurtej K. Dhoot(University of London), J. Paul Chapple(Queen Mary University of London), Anne Hamechar-Brady(Johns Hopkins University), Roland A. Fleck(King's College London), Daniel A. East(University of London), György Szabadkai(University College London), Laura Kuhlman Hussey(University of London), Rosella Abeti(National Hospital for Neurology and Neurosurgery), Aarti Singh(University of London), Gema Vizcay‐Barrena(King's College London)
Cited by 3
Related Papers
Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018
|Cell Death and Differentiation|2018|6.6k
Chaperone-mediated coupling of endoplasmic reticulum and mitochondrial Ca2+ channels
|The Journal of Cell Biology|2006|1.4k
Highly Pathogenic Avian Influenza A(H5N1) Clade 2.3.4.4b Virus Infection in Domestic Dairy Cattle and Cats, United States, 2024
|Emerging infectious diseases|2024|535
Apoptotic cell death in disease—Current understanding of the NCCD 2023
|Cell Death and Differentiation|2023|371
XBP1 mRNA Splicing Triggers an Autophagic Response in Endothelial Cells through BECLIN-1 Transcriptional Activation
|Journal of Biological Chemistry|2012|277