E6-mediated activation of JNK drives EGFR signalling to promote proliferation and viral oncoprotein expression in cervical cancerEthan L. Morgan(University of Leeds), Andrew Macdonald(University of Leeds)Cell Death and DifferentiationDecember 10, 202010.1038/s41418-020-00693-9Cited by 100SaveCiteExport RISWatch citationsRelated PapersSTAT3 activation by E6 is essential for the differentiation-dependent HPV18 life cycle|PLoS Pathogens|2018|92Human papillomavirus type 18 E5 oncogene supports cell cycle progression and impairs epithelial differentiation by modulating growth factor receptor signalling during the virus life cycle|Oncotarget|2017|72The deubiquitinase (DUB) USP13 promotes Mcl-1 stabilisation in cervical cancer|Oncogene|2021|56The PP4R1 sub-unit of protein phosphatase PP4 is essential for inhibition of NF-κB by merkel polyomavirus small tumour antigen|Oncotarget|2017|46Exploitation of ATP-sensitive potassium ion (KATP) channels by HPV promotes cervical cancer cell proliferation by contributing to MAPK/AP-1 signalling|Oncogene|2023|20