SUCLA2 mutations cause global protein succinylation contributing to the pathomechanism of a hereditary mitochondrial disease

Philipp Gut(University of California, San Francisco), Eric Verdin(Buck Institute for Research on Aging), Christopher J. Carroll(St George's, University of London), Pirjo Isohanni(University of Helsinki), Matthew J. Rardin(Amgen (United States)), Christopher B. Jackson(Memorial Sloan Kettering Cancer Center), Stefan Christen(Nestlé (Switzerland)), Yuya Nishida(Gladstone Institutes), Sofia Moco(Centre for BioSystems Genomics), Pieti W. Pallijeff(University of Helsinki), Reem A. Alkhater(Saudi Aramco Medical Services Organization), Jason W. Locasale(Duke University), Liliya Euro(University of Helsinki), Anu Suomalainen(University of Helsinki), Elsebet Østergaard(Copenhagen University Hospital), Jesse G. Meyer(Cedars-Sinai Medical Center), Gabriele Civiletto(University of Cambridge), Sanna Matilainen(University of Helsinki), John C. Newman(Buck Institute for Research on Aging), J. Wall Richard(Nestlé (Switzerland)), Xiaojing Liu(Duke University), Wenjuan He(Gladstone Institutes), Alice Parisi(University of Verona), Jonathan Thévenet(Nestlé (Switzerland)), Berge A. Minassian(Southwestern Medical Center), Birgit Schilling(Buck Institute for Research on Aging)
Nature Communications
November 23, 2020
Cited by 82


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