Mitochondrial dysfunction underlying sporadic inclusion body myositis is ameliorated by the mitochondrial homing drug MA-5
Yoshitsugu Oikawa(Tohoku University), Takaaki Abe(Tohoku University)
Cited by 36
Related Papers
Mutation of the Mg2+ Transporter SLC41A1 Results in a Nephronophthisis-Like Phenotype
|Journal of the American Society of Nephrology|2013|71
Mitochonic Acid 5 (MA-5) Facilitates ATP Synthase Oligomerization and Cell Survival in Various Mitochondrial Diseases
|EBioMedicine|2017|68
A novel indole compound MA-35 attenuates renal fibrosis by inhibiting both TNF-α and TGF-β1 pathways
|Scientific Reports|2017|30
Transcriptomic Hallmarks of Mortality Reveal Universal and Specific Mechanisms of Aging, Chronic Disease, and Rejuvenation
|bioRxiv (Cold Spring Harbor Laboratory)|2024|26
Urinary growth differentiation factor 15 predicts renal function decline in diabetic kidney disease
|Scientific Reports|2023|16