Mitochondria form contact sites with the nucleus to couple pro-survival retrograde response
Radha Desai(University of London), Michelangelo Campanella(Centre National de la Recherche Scientifique), Valentina Zappulli(University of Padua), María Soledad Álvarez(University of London), Lisa E. L. Romano(Queen Mary University of London), Robert B. Bentham(MRC Laboratory for Molecular Cell Biology), Liana Hardy(University of London), James Crosby(University of London), Danilo Faccenda(University of London), Ken Smith(University of Utah), Isabelle Coppens(Johns Hopkins University), Marta Mainenti(Iowa State University), Gurtej K. Dhoot(University of London), Roland A. Fleck(King's College London), Daniel A. East(University of London), György Szabadkai(University College London), Laura Kuhlman Hussey(University of London), Rosella Abeti(National Hospital for Neurology and Neurosurgery), Aarti Singh(University of London), Manuel Rigon(William Harvey Research Institute)
UCL Discovery (University College London)
December 1, 2020
Cited by 14
Related Papers
Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018
|Cell Death and Differentiation|2018|6.6k
Chaperone-mediated coupling of endoplasmic reticulum and mitochondrial Ca2+ channels
|The Journal of Cell Biology|2006|1.4k
Highly Pathogenic Avian Influenza A(H5N1) Clade 2.3.4.4b Virus Infection in Domestic Dairy Cattle and Cats, United States, 2024
|Emerging infectious diseases|2024|535
Apoptotic cell death in disease—Current understanding of the NCCD 2023
|Cell Death and Differentiation|2023|371
<i>Toxoplasma gondii</i>Exploits Host Low-Density Lipoprotein Receptor-Mediated Endocytosis for Cholesterol Acquisition
|The Journal of Cell Biology|2000|300