Gut microbiota–specific IgA <sup>+</sup> B cells traffic to the CNS in active multiple sclerosis
Anne‐Katrin Pröbstel(University of California, San Francisco), Xiaoyuan Zhou(University of California, San Francisco), Ryan Baumann(University of California, San Francisco), Sven Wischnewski(University of Mannheim), Michael Kutza(University of Mannheim), Olga L. Rojas(University of Toronto), Katrin Sellrie(Max Planck Institute of Colloids and Interfaces), Antje Bischof(University of California, San Francisco), Kicheol Kim(University of California, San Francisco), Akshaya Ramesh(University of California, San Francisco), Ravi Dandekar(University of California, San Francisco), Ariele L. Greenfield(University of California, San Francisco), Ryan Schubert(University of California, San Francisco), Jordan E. Bisanz(University of California, San Francisco), Stephanie Vistnes(Broad Center), Khashayar Khaleghi(University of Toronto), James Landefeld(University of California, San Francisco), Gina Kirkish(University of California, San Francisco), Friederike Liesche‐Starnecker(Technical University of Munich), Valeria Ramaglia(University of Mannheim), Sneha Singh(University of California, San Francisco), Edwina B. Tran(University of California, San Francisco), Patrick Barba(University of California, San Francisco), Kelsey C. Zorn(University of California, San Francisco), Johanna Oechtering(University Hospital of Basel), Karin Forsberg(Umeå University), Lawrence R. Shiow(University of California, San Francisco), Roland G. Henry(University of California, San Francisco), Jennifer Graves(University of California, San Francisco), Bruce Cree(University of California, San Francisco), Stephen L. Hauser(University of California, San Francisco), Jens Kühle(University Hospital of Basel), Jeffrey M. Gelfand(University of California, San Francisco), Peter M. Andersen(Umeå University), Jürgen Schlegel(Technical University of Munich), Peter J. Turnbaugh(University of California, San Francisco), Peter H. Seeberger(Max Planck Institute of Colloids and Interfaces), Jennifer L. Gommerman(University of Toronto), Michael R. Wilson(University of California, San Francisco), Lucas Schirmer(University of Mannheim), Sergio E. Baranzini(University of California, San Francisco)
Cited by 219Open Access
Abstract
cells as a systemic mediator in MS and suggest a critical role of mucosal B cells during active neuroinflammation with broad implications for IgA as an informative biomarker and IgA-producing cells as an immune subset to harness for therapeutic interventions.
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