Targeted deletions of complement lectin pathway genes improve outcome in traumatic brain injury, with MASP-2 playing a major role
Domenico Mercurio(Mario Negri Institute for Pharmacological Research), Maria Grazia De Simoni(Istituto di Fisiologia Clinica), Wilhelm Schwaeble(University of Cambridge), Nicholas J. Lynch(Mario Negri Institute for Pharmacological Research), Russell Wallis(University of Leeds), Silke Roscher(University of Leicester), Stefano Fumagalli(Mario Negri Institute for Pharmacological Research), Stefania Ippati(Mario Negri Institute for Pharmacological Research), Denise Minuta(Vita-Salute San Raffaele University), Marco Oggioni(Mario Negri Institute for Pharmacological Research), Carlo Perego(Mario Negri Institute for Pharmacological Research)
Cited by 26
Related Papers
Interleukin-1β Immunoreactivity and Microglia Are Enhanced in the Rat Hippocampus by Focal Kainate Application: Functional Evidence for Enhancement of Electrographic Seizures
|Journal of Neuroscience|1999|567
Temporal pattern of expression and colocalization of microglia/macrophage phenotype markers following brain ischemic injury in mice
|Journal of Neuroinflammation|2011|517
Inflammatory cytokines and related genes are induced in the rat hippocampus by limbic status epilepticus
|European Journal of Neuroscience|2000|513
Functional Role of Inflammatory Cytokines and Antiinflammatory Molecules in Seizures and Epileptogenesis
|Epilepsia|2002|385
Site-Directed Conjugation of “Clicked” Glycopolymers To Form Glycoprotein Mimics: Binding to Mammalian Lectin and Induction of Immunological Function
|Journal of the American Chemical Society|2007|299