Active Akt signaling triggers CLL toward Richter transformation via overactivation of Notch1
Vivien Kohlhaas(University of Cologne), F. Thomas Wunderlich(University of Cologne), Mona Al-Maarri(University of Cologne), Reinhard Büttner(University Hospital Cologne), Marcus Krüger(Cologne Excellence Cluster on Cellular Stress Responses in Aging Associated Diseases), Davide Rossi(Università degli Studi del Piemonte Orientale “Amedeo Avogadro”), Renzo Boldorini(Università degli Studi del Piemonte Orientale “Amedeo Avogadro”), Miloš Nikolić(Genomics (United Kingdom)), Janica L. Wiederstein(Cologne Excellence Cluster on Cellular Stress Responses in Aging Associated Diseases), Riccardo Moia(Università degli Studi del Piemonte Orientale “Amedeo Avogadro”), Andreas Rosenwald(Comprehensive Cancer Center Mainfranken), Carmen Herling(University Hospital Leipzig), Hans Christian Reinhardt(German Cancer Research Center), Stuart J. Blakemore(University of Southampton), Marco Herling(Unknown), Gianluca Gaïdano(Università degli Studi del Piemonte Orientale “Amedeo Avogadro”), Gero Knittel(University of Cologne), Christian P. Pallasch(University of Cologne), Nadine Nickel(University of Cologne), Nadine Hövelmeyer(Johannes Gutenberg University Mainz), Elena Hartmann(University of Würzburg), Martin Pál(Charles Sturt University), Lukas P. Frenzel(University of Cologne), Martin Peifer(University of Cologne), Stephan Schäfer(Taipei Institute of Pathology), Nina Reinart(University Hospital Cologne), Michael Hallek(Integrated Oncology (United States)), Marek Franitza(Cologne Excellence Cluster on Cellular Stress Responses in Aging Associated Diseases), Jens C. Brüning(University of Cologne), Andreas Röth(Helios Klinikum Emil von Behring), Theodoros Georgomonolis(University of Cologne), Philipp Lohneis(Taipei Institute of Pathology)
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