Patient-Specific Induced Pluripotent Stem Cells Implicate Intrinsic Impaired Contractility in Hypoplastic Left Heart Syndrome
Sharon L. Paige(Cytokinetics (United States)), Sean M. Wu(Cardiovascular Institute of the South), Soah Lee(Sungkyunkwan University), Francisco X. Galdos(Cardiovascular Institute of the South), Dries Feyen(MACOM (United States)), Aimee Beck(Cardiovascular Institute of the South), Mark Mercola(Cardiovascular Institute of the South), Marlene Rabinovitch(Stanford Medicine), Sara Ranjbarvaziri(Unknown), Elizabeth T. Chin(Johns Hopkins University), Mingxia Gu(Community Health Center), Yifei Miao(Donghua University), Adrija Darsha(MACOM (United States)), Victoria N. Parikh(Stanford University), Daniel Bernstein(Stanford University), M. Yasir Qureshi(Mayo Clinic in Arizona), Euan A. Ashley(Stanford University), Sidra Xu(MACOM (United States)), Timothy J. Nelson(Mayo Clinic), Julia Ryan(MACOM (United States))
Cited by 54
Related Papers
Autophagy proteins regulate innate immune responses by inhibiting the release of mitochondrial DNA mediated by the NALP3 inflammasome
|Nature Immunology|2010|3k
Genotype and Lifetime Burden of Disease in Hypertrophic Cardiomyopathy
|Circulation|2018|837
Rat pulmonary circulation after chronic hypoxia: hemodynamic and structural features
|American Journal of Physiology-Heart and Circulatory Physiology|1979|484
Wnt antagonism initiates cardiogenesis in <i>Xenopus laevis</i>
|Genes & Development|2001|482