B cells polarize pathogenic inflammatory T helper subsets through ICOSL-dependent glycolysis
Qiu‐Hui Zeng(Sun Yat-sen University), Wei Yuan(Sun Yat-sen University), Xiang‐Ming Lao(Sun Yat-sen University), Dong‐Ping Chen(Sun Yat-sen University), Chun-Xiang Huang(Sun Yat-sen University), Qianyi Lin(Sun Yat-sen University), Min He(Guangzhou University of Chinese Medicine), Yuan Liao(Sun Yat-sen University), Limin Zheng(Sun Yat-sen University), Bo Li(Sun Yat-sen University), Guangbo Zhang(Soochow University), Yun Chen(Nanjing Medical University), Dong‐Ming Kuang(Sun Yat-sen University)
Cited by 37Open Access
Abstract
B cells have a previously unrecognized helper function in triggering pathogenic differentiation of inflammatory T H subsets.
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