The deubiquitinase USP44 promotes Treg function during inflammation by preventing FOXP3 degradation
Jing Yang(Yangzhou Polytechnic Institute), Bin Li(Shanghai Jiao Tong University), Chichu Xie(Sun Yat-sen University), Wenzhi Guo(First Affiliated Hospital of Zhengzhou University), Paul J. Galardy(Mayo Clinic in Arizona), Guochao Shi(Shanghai Jiao Tong University), Jin‐Hui Tao(University of Science and Technology of China), Juan Fu(Henan Forestry Vocational College), Xuemei Tong(Shanghai Jiao Tong University), Yakun Bai(First Affiliated Hospital of Zhengzhou University), Ying Lu(Stanford University), Xiaoxia Hou(Shanghai Jiao Tong University), Yanhang Gao(Jilin University), Zuojia Chen(Brigham and Women's Hospital), Evan Yang(Johns Hopkins University), Shuijun Zhang(Open University of China), Joseph Barbi(Johns Hopkins University), Dan Li(Harbin Medical University), Ying Zhang(Air Force Medical University), Songguo Zheng(Penn State Milton S. Hershey Medical Center), Huabin Li(Guangxi Zhuang Autonomous Region Health and Family Planning), Ping Wei(Shanghai Medical College of Fudan University), Jia Nie(Shanghai Institutes for Biological Sciences), Jian Meng(Shanghai Jiao Tong University), Fan Pan(Nanjing General Hospital of Nanjing Military Command), Xingmei Wu(Shanghai University of Traditional Chinese Medicine), Rui Liang(Nankai University), Wei Kou(China International Science and Technology Cooperation), Andy Tsun(Shanghai Jiao Tong University), Jiazi Ren(Institut Pasteur of Shanghai), Junqi Niu(Jilin University), Qianru Huang(Shanghai Jiao Tong University), Yayi Gao(Institut Pasteur of Shanghai)
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