Expression of Annexin A2 Promotes Cancer Progression in Estrogen Receptor Negative Breast Cancers

Amira F. Mahdi(University of Limerick), Beatrice Malacrida(University of Limerick), Joanne Nolan(University of Limerick), Mary McCumiskey(University of Limerick), Anne Merrigan(University Hospital Limerick), Ashish Lal(University Hospital Limerick), Shona Tormey(University Hospital Limerick), Aoïfe Lowery(Ollscoil na Gaillimhe – University of Galway), Kieran McGourty(University of Limerick), Patrick A. Kiely(University of Limerick)
Cells
June 30, 2020
Cited by 22Open Access
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Abstract

When breast cancer progresses to a metastatic stage, survival rates decline rapidly and it is considered incurable. Thus, deciphering the critical mechanisms of metastasis is of vital importance to develop new treatment options. We hypothesize that studying the proteins that are newly synthesized during the metastatic processes of migration and invasion will greatly enhance our understanding of breast cancer progression. We conducted a mass spectrometry screen following bioorthogonal noncanonical amino acid tagging to elucidate changes in the nascent proteome that occur during epidermal growth factor stimulation in migrating and invading cells. Annexin A2 was identified in this screen and subsequent examination of breast cancer cell lines revealed that Annexin A2 is specifically upregulated in estrogen receptor negative (ER-) cell lines. Furthermore, siRNA knockdown showed that Annexin A2 expression promotes the proliferation, wound healing and directional migration of breast cancer cells. In patients, Annexin A2 expression is increased in ER- breast cancer subtypes. Additionally, high Annexin A2 expression confers a higher probability of distant metastasis specifically for ER- patients. This work establishes a pivotal role of Annexin A2 in breast cancer progression and identifies Annexin A2 as a potential therapeutic target for the more aggressive and harder to treat ER- subtype.


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