Excess Rab4 rescues synaptic and behavioral dysfunction caused by defective HTT-Rab4 axonal transport in Huntington’s diseaseJoseph White(Florida International University), Shermali Gunawardena(Buffalo State University)Acta Neuropathologica CommunicationsJuly 1, 202010.1186/s40478-020-00964-zCited by 37SaveCiteExport RISWatch citationsRelated PapersA New Set of Arabidopsis Expressed Sequence Tags from Developing Seeds. The Metabolic Pathway from Carbohydrates to Seed Oil,|PLANT PHYSIOLOGY|2000|238HTT (huntingtin) and RAB7 co-migrate retrogradely on a signaling LAMP1-containing late endosome during axonal injury|Autophagy|2022|15Opposing roles for GSK3β and ERK1-dependent phosphorylation of huntingtin during neuronal dysfunction and cell death in Huntington’s disease|Cell Death and Disease|2025|4