17-Aminogeldanamycin Inhibits Constitutive Nuclear Factor-Kappa B (NF-κB) Activity in Patient-Derived Melanoma Cell Lines

Mariusz L. Hartman(Medical University of Lodz), Małgorzata Czyż(Medical University of Lodz)

International Journal of Molecular Sciences

May 26, 2020

10.3390/ijms21113749

Cited by 17

Related Papers

Gene Expression Profiling Identifies Microphthalmia-Associated Transcription Factor (MITF) and Dickkopf-1 (DKK1) as Regulators of Microenvironment-Driven Alterations in Melanoma Phenotype

|PLoS ONE|2014|33

Vemurafenib and trametinib reduce expression of CTGF and IL-8 in V600EBRAF melanoma cells

|Laboratory Investigation|2017|32

Physiologically Relevant Oxygen Concentration (6% O2) as an Important Component of the Microenvironment Impacting Melanoma Phenotype and Melanoma Response to Targeted Therapeutics In Vitro

|International Journal of Molecular Sciences|2019|24

17-Aminogeldanamycin selectively diminishes IRE1α-XBP1s pathway activity and cooperatively induces apoptosis with MEK1/2 and BRAFV600E inhibitors in melanoma cells of different genetic subtypes

|APOPTOSIS|2019|20

Pentoxifylline Inhibits WNT Signalling in β-Cateninhigh Patient-Derived Melanoma Cell Populations

|PLoS ONE|2016|15