Reduced contractile reserve and diastolic dysfunction in a trangenic mouse model with beta(1)-adrenergic receptor overexpression assessed by MRI

Frank Wiesmann(University of Würzburg), Stefan Neubauer(John Radcliffe Hospital), CO Ritter, Stefan Engelhardt(Heidelberg University), Lutz Hein(University of Freiburg), Axel Haase(University of Würzburg), Martin J. Lohse(University of Würzburg), Jan Ruff(University of Würzburg)
Circulation
November 2, 1999
Cited by 7

Related Papers

MicroRNA-21 contributes to myocardial disease by stimulating MAP kinase signalling in fibroblasts
|Nature|2008|2.4k
Non-invasive detection of coronary inflammation using computed tomography and prediction of residual cardiovascular risk (the CRISP CT study): a post-hoc analysis of prospective outcome data
|The Lancet|2018|1.1k
Cardiac fibroblast–derived microRNA passenger strand-enriched exosomes mediate cardiomyocyte hypertrophy
|Journal of Clinical Investigation|2014|993
MicroRNAs in the Human Heart
|Circulation|2007|888
Progressive hypertrophy and heart failure in β <sub>1</sub> -adrenergic receptor transgenic mice
|Proceedings of the National Academy of Sciences|1999|787