Caspase-8-Dependent Inflammatory Responses Are Controlled by Its Adaptor, FADD, and Necroptosis
Bart Tummers(King's College London), Douglas R. Green(St. Jude Children's Research Hospital), J. Magarian Blander(Tisch Hospital), Luigi Mari(St. Jude Children's Research Hospital), Jeremy Chase Crawford(St. Jude Children's Research Hospital), Bradlee L. Heckmann(St. Jude Children's Research Hospital), Julien Moretti(Cornell University), Thirumala‐Devi Kanneganti(St. Jude Children's Research Hospital), Laura J. Janke(St. Jude Children's Research Hospital), Diego A. Rodríguez(St. Jude Children's Research Hospital), Patrick Fitzgerald(University College Cork), Sebastian Rühl(St. Jude Children's Research Hospital), Clifford S. Guy(St. Jude Children's Research Hospital), S. William Pelletier(Indiana University School of Medicine)
Cited by 174
Related Papers
Classification of cell death: recommendations of the Nomenclature Committee on Cell Death 2009
|Cell Death and Differentiation|2008|3.3k
Early redistribution of plasma membrane phosphatidylserine is a general feature of apoptosis regardless of the initiating stimulus: inhibition by overexpression of Bcl-2 and Abl.
|The Journal of Experimental Medicine|1995|2.9k
The microbiome-gut-brain axis during early life regulates the hippocampal serotonergic system in a sex-dependent manner
|Molecular Psychiatry|2012|1.9k
HIF1α–dependent glycolytic pathway orchestrates a metabolic checkpoint for the differentiation of TH17 and Treg cells
|The Journal of Experimental Medicine|2011|1.8k
Transferring the blues: Depression-associated gut microbiota induces neurobehavioural changes in the rat
|Journal of Psychiatric Research|2016|1.7k