Postnatal Ethanol-Induced Neurodegeneration Involves CB1R-Mediated β-Catenin Degradation in Neonatal Mice
Shivakumar Subbanna(Nathan Kline Institute for Psychiatric Research), Balapal S. Basavarajappa(Nathan Kline Institute for Psychiatric Research)
Cited by 7
Related Papers
Anandamide–CB<sub>1</sub>Receptor Signaling Contributes to Postnatal Ethanol-Induced Neonatal Neurodegeneration, Adult Synaptic, and Memory Deficits
|Journal of Neuroscience|2013|103
Ethanol Exposure Induces Neonatal Neurodegeneration by Enhancing CB1R Exon1 Histone H4K8 Acetylation and Up-regulating CB1R Function causing Neurobehavioral Abnormalities in Adult Mice
|The International Journal of Neuropsychopharmacology|2014|73
CB1R-Mediated Activation of Caspase-3 Causes Epigenetic and Neurobehavioral Abnormalities in Postnatal Ethanol-Exposed Mice
|Frontiers in Molecular Neuroscience|2018|29