Ubiquitinated-PCNA protects replication forks from DNA2-mediated degradation by regulating Okazaki fragment maturation and chromatin assembly
Tanay Thakar(Pennsylvania State University), George‐Lucian Moldovan(Pennsylvania State University)
Cited by 138
Related Papers
Loss of E2F7 confers resistance to poly-ADP-ribose polymerase (PARP) inhibitors in BRCA2-deficient cells
|Nucleic Acids Research|2018|71
HUWE1 interacts with PCNA to alleviate replication stress
|EMBO Reports|2016|65
Identification of regulators of poly-ADP-ribose polymerase inhibitor response through complementary CRISPR knockout and activation screens
|Nature Communications|2020|52
The KU-PARP14 axis differentially regulates DNA resection at stalled replication forks by MRE11 and EXO1
|Nature Communications|2022|47
Multi-step processing of replication stress-derived nascent strand DNA gaps by MRE11 and EXO1 nucleases
|Nature Communications|2023|46