CRISPRi-based radiation modifier screen identifies long non-coding RNA therapeutic targets in glioma

Siyuan Liu(San Francisco VA Medical Center), Martina Malatesta(San Francisco VA Medical Center), Brian V. Lien(San Francisco VA Medical Center), Parna Saha(San Francisco VA Medical Center), Shivani S. Thombare(San Francisco VA Medical Center), Sung Jun Hong(San Francisco VA Medical Center), Leslie Pedraza(San Francisco VA Medical Center), Mark Koontz(University of California, San Francisco), Kyounghee Seo(University of California, San Francisco), Max A. Horlbeck(QB3), Daniel He(San Francisco VA Medical Center), Harjus Birk(San Francisco VA Medical Center), Miten Jain(University of California, Santa Cruz), Hugh E. Olsen(University of California, Santa Cruz), Mark Akeson(University of California, Santa Cruz), Jonathan S. Weissman(QB3), Michelle Monje(Stanford University), Nalin Gupta(University of California, San Francisco), David R. Raleigh(University of California, San Francisco), Erik M. Ullian(University of California, San Francisco), Daniel A. Lim(San Francisco VA Medical Center)
Genome biology
March 31, 2020
Cited by 154Open Access
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Abstract

BACKGROUND: Long non-coding RNAs (lncRNAs) exhibit highly cell type-specific expression and function, making this class of transcript attractive for targeted cancer therapy. However, the vast majority of lncRNAs have not been tested as potential therapeutic targets, particularly in the context of currently used cancer treatments. Malignant glioma is rapidly fatal, and ionizing radiation is part of the current standard-of-care used to slow tumor growth in both adult and pediatric patients. RESULTS: We use CRISPR interference (CRISPRi) to screen 5689 lncRNA loci in human glioblastoma (GBM) cells, identifying 467 hits that modify cell growth in the presence of clinically relevant doses of fractionated radiation. Thirty-three of these lncRNA hits sensitize cells to radiation, and based on their expression in adult and pediatric gliomas, nine of these hits are prioritized as lncRNA Glioma Radiation Sensitizers (lncGRS). Knockdown of lncGRS-1, a primate-conserved, nuclear-enriched lncRNA, inhibits the growth and proliferation of primary adult and pediatric glioma cells, but not the viability of normal brain cells. Using human brain organoids comprised of mature neural cell types as a three-dimensional tissue substrate to model the invasive growth of glioma, we find that antisense oligonucleotides targeting lncGRS-1 selectively decrease tumor growth and sensitize glioma cells to radiation therapy. CONCLUSIONS: These studies identify lncGRS-1 as a glioma-specific therapeutic target and establish a generalizable approach to rapidly identify novel therapeutic targets in the vast non-coding genome to enhance radiation therapy.


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