Evolution of tumor cells during AsiDNA treatment results in energy exhaustion, decrease in responsiveness to signal, and higher sensitivity to the drug

Pierre‐Marie Girard(Université Paris Sciences et Lettres), Marie Dutreix(Institut Curie)
Evolutionary Applications
March 10, 2020
10.1111/eva.12949
Cited by 6

Related Papers

Hyperactivation of DNA-PK by Double-Strand Break Mimicking Molecules Disorganizes DNA Damage Response
|PLoS ONE|2009|80
Singlet Oxygen-Mediated Oxidation during UVA Radiation Alters the Dynamic of Genomic DNA Replication
|PLoS ONE|2015|34
The AsiDNA™ decoy mimicking DSBs protects the normal tissue from radiation toxicity through a DNA-PK/p53/p21-dependent G1/S arrest
|NAR Cancer|2024|8