Heart failure after pressure overload in autosomal-dominant desminopathies: Lessons from heterozygous DES-p.R349P knock-in mice
Florian Stöckigt(University Hospital Bonn), Jan W. Schrickel(University Hospital Bonn), Lisa Kamm(Friedrich-Alexander-Universität Erlangen-Nürnberg), Georg Nickenig(University of Bonn), Vincent Knappe(University Hospital Bonn), Ursula Schlötzer‐Schrehardt(Friedrich-Alexander-Universität Erlangen-Nürnberg), Christoph S. Clemen(University of Cologne), Tobias Radecke, Martin Steinmetz, Thomas Beiert(University Hospital Bonn), Carolin Berwanger, Dorothea Schultheis(Friedrich-Alexander-Universität Erlangen-Nürnberg), Viktoriya Peeva(University of Bonn), Wolfram S. Kunz(Klinikum Magdeburg), Rolf Schröder(Friedrich-Alexander-Universität Erlangen-Nürnberg), Alexei P. Kudin, Lars Eichhorn(University Hospital Bonn)
Cited by 5
Related Papers
Increased NADH-Oxidase–Mediated Superoxide Production in the Early Stages of Atherosclerosis
|Circulation|1999|671
Evaluating Real-World Clinical Outcomes in Atrial Fibrillation Patients Receiving the WATCHMAN Left Atrial Appendage Closure Technology
|Circulation Arrhythmia and Electrophysiology|2019|286
Right Ventricular-Pulmonary Arterial Coupling and Afterload Reserve in Patients Undergoing Transcatheter Tricuspid Valve Repair
|Journal of the American College of Cardiology|2022|261
Loss-of-function mutations in MGME1 impair mtDNA replication and cause multisystemic mitochondrial disease
|Nature Genetics|2013|237
Linear mitochondrial DNA is rapidly degraded by components of the replication machinery
|Nature Communications|2018|224