Synaptic mitochondrial dysfunction and septin accumulation are linked to complement-mediated synapse loss in an Alzheimer’s disease animal model
Balázs A. Györffy(University of Copenhagen), József Kardos(Eötvös Loránd University), Miklós Sántha(HUN-REN Szegedi Biológiai Kutatóközpont), Péter Gulyássy(Hungarian Academy of Sciences), Vilmos Tóth(Eötvös Loránd University), Melinda Tóth(HUN-REN Szegedi Biológiai Kutatóközpont), László Homolya(HUN-REN Research Centre for Natural Sciences), András Micsonai(Eötvös Loránd University), Katalin A. Kékesi(Eötvös Loránd University), Henrietta Vadászi(Eötvös Loránd University), Gábor Juhász(Eötvös Loránd University), Réka Á. Kovács(Eötvös Loránd University), György Török(Institute of Molecular Life Sciences), László Drahos(Hungarian Academy of Sciences)
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