N6-Isopentenyladenosine Enhances the Radiosensitivity of Glioblastoma Cells by Inhibiting the Homologous Recombination Repair Protein RAD51 Expression

Giovanna Navarra(University of Naples Federico II), Maurizio Bifulco(University of Naples Federico II)

Frontiers in Oncology

January 13, 2020

10.3389/fonc.2019.01498

Cited by 15

Related Papers

Cannabinoid receptor CB1 regulates STAT3 activity and its expression dictates the responsiveness to SR141716 treatment in human glioma patients' cells

|Oncotarget|2015|54

Antiglioma effects of<scp>N</scp>6‐isopentenyladenosine, an endogenous isoprenoid end product, through the downregulation of epidermal growth factor receptor

|International Journal of Cancer|2016|34

The isoprenoid end product N6-isopentenyladenosine reduces inflammatory response through the inhibition of the NFκB and STAT3 pathways in cystic fibrosis cells

|Inflammation Research|2017|14

<scp>N6</scp>‐isopentenyladenosine inhibits aerobic glycolysis in glioblastoma cells by targeting <scp>PKM2</scp> expression and activity

|FEBS Open Bio|2024|6