Succinate dehydrogenase deficiency in a chromaffin cell model retains metabolic fitness through the maintenance of mitochondrial NADH oxidoreductase function
Katarína Kľučková(The Edgbaston Hospital), Daniel A. Tennant(University of Birmingham), Jacqueline L.E. Tearle(University of Birmingham), Judith Favier(Inserm), Cristina Escribano-Gonzalez(University of Birmingham), Alpesh Thakker(The Edgbaston Hospital), Baksho Kaul(University of Birmingham), Gareth G. Lavery(University of Birmingham), Rebecca L. Hindshaw(University of Birmingham), Lisa A. Vettore(The Edgbaston Hospital), Judith Goncalves(Stanford University)
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