TNF-a Induces a Pro-Inflammatory Phenotypic Shift in Monocytes through ACSL1: Relevance to Metabolic Inflammation

Fatema Al‐Rashed(Mubarak Al Kabeer Hospital), Rasheed Ahmad(Dasman Diabetes Institute)
Cellular Physiology and Biochemistry
March 7, 2019
10.33594/000000028
Cited by 47

Related Papers

The Synergy between Palmitate and TNF-α for CCL2 Production Is Dependent on the TRIF/IRF3 Pathway: Implications for Metabolic Inflammation
|The Journal of Immunology|2018|92
Palmitate-Induced MMP-9 Expression in the Human Monocytic Cells is Mediated through the TLR4-MyD88 Dependent Mechanism
|Cellular Physiology and Biochemistry|2016|73
Neutral sphingomyelinase 2 regulates inflammatory responses in monocytes/macrophages induced by TNF-α
|Scientific Reports|2020|63
Repetitive Intermittent Hyperglycemia Drives the M1 Polarization and Inflammatory Responses in THP-1 Macrophages Through the Mechanism Involving the TLR4-IRF5 Pathway
|Cells|2020|60
FSL-1 Induces MMP-9 Production through TLR-2 and NF-κB /AP-1 Signaling Pathways in Monocytic THP-1 Cells
|Cellular Physiology and Biochemistry|2014|57