GLP-catalyzed H4K16me1 promotes 53BP1 recruitment to permit DNA damage repair and cell survival
Xiaopeng Lu(Shenzhen Metro (China)), Wei‐Guo Zhu(Shenzhen University), Yafei Lv(Shenzhen University), Qian Zhu(East China University of Science and Technology), Zhongyi Cheng(New England Biolabs (China)), Luo Gu(Johns Hopkins University), Xingzhi Xu(Northwest Institute of Eco-Environment and Resources), Qiaoyan Yang(Shenzhen University), Haiying Wang(Southern Medical University), Wen He(Beijing Chemical Industry Research Institute (China)), Baohua Liu(Shenzhen University), Ming Tang(Shanghai First Maternity and Infant Hospital), Zhiming Li(Shenzhen University), Ge Liu(Shenzhen University), Yantao Bao(Shenzhen University), Tianyun Hou(Peking University), Yang Yang(First Affiliated Hospital of Zhengzhou University), Ying Zhao(Zhejiang Chinese Medical University)
Cited by 42
Related Papers
Genomic instability in laminopathy-based premature aging
|Nature Medicine|2005|641
SoNar, a Highly Responsive NAD+/NADH Sensor, Allows High-Throughput Metabolic Screening of Anti-tumor Agents
|Cell Metabolism|2015|428
The Roles of Histone Deacetylases and Their Inhibitors in Cancer Therapy
|Frontiers in Cell and Developmental Biology|2020|362
Lateral Sharpening of Cortical Frequency Tuning by Approximately Balanced Inhibition
|Neuron|2008|291
Visual Representations by Cortical Somatostatin Inhibitory Neurons—Selective But with Weak and Delayed Responses
|Journal of Neuroscience|2010|244