Glycogen synthase kinase 3β hyperactivity in urinary exfoliated cells predicts progression of diabetic kidney disease
Xianhui Liang(First Affiliated Hospital of Zhengzhou University), Rujun Gong(Brown University)
Cited by 56
Related Papers
Hepatocyte Growth Factor Suppresses Proinflammatory NFκB Activation through GSK3β Inactivation in Renal Tubular Epithelial Cells
|Journal of Biological Chemistry|2008|97
Hepatocyte growth factor suppresses acute renal inflammation by inhibition of endothelial E-selectin
|Kidney International|2006|55
Permissive effect of GSK3β on profibrogenic plasticity of renal tubular cells in progressive chronic kidney disease
|Cell Death and Disease|2021|47
Rescue therapy with Tanshinone IIA hinders transition of acute kidney injury to chronic kidney disease via targeting GSK3β
|Scientific Reports|2016|46
Glycogen Synthase Kinase 3β: A Novel Marker and Modulator of Inflammatory Injury in Chronic Renal Allograft Disease
|American Journal of Transplantation|2008|43