Parkin‐mediated ubiquitylation redistributes MITOL/March5 from mitochondria to peroxisomes
Fumika Koyano(Tokyo Medical and Dental University), Noriyuki Matsuda(Tokyo Medical and Dental University), Hidetaka Kosako(Institute for Advanced Medical Research), Yukio Fujiki(Kyushu University), Yoko Kimura(Tokyo Metropolitan Institute of Medical Science), Mayumi Kimura(Tokyo Metropolitan Institute of Medical Science), Koji Yamano(Tokyo Metropolitan Institute of Medical Science), Keiji Tanaka(Tokyo Metropolitan Institute of Medical Science)
Cited by 60
Related Papers
Autophagy regulates lipid metabolism
|Nature|2009|3.9k
Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase
|Nature Genetics|2000|2k
PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy
|The Journal of Cell Biology|2010|1.9k
Ubiquitin is phosphorylated by PINK1 to activate parkin
|Nature|2014|1.5k
The proteasome: Overview of structure and functions
|Proceedings of the Japan Academy Series B|2009|860