Rebalancing of actomyosin contractility enables mammary tumor formation upon loss of E-cadherin
Koen Schipper(The Netherlands Cancer Institute), Jos Jonkers(The Netherlands Cancer Institute), Arnoud Sonnenberg(The Netherlands Cancer Institute), Micha Nethe(Amsterdam UMC Location University of Amsterdam), Jacco van Rheenen(The Netherlands Cancer Institute), Eline van der Burg(The Netherlands Cancer Institute), Veronika Ramovš(Leiden University Medical Center), Daniëlle Seinstra(The Netherlands Cancer Institute), Anne Paulien Drenth(The Netherlands Cancer Institute)
Cited by 36
Related Papers
Function and interactions of integrins
|Cell and Tissue Research|2001|1k
53BP1 loss rescues BRCA1 deficiency and is associated with triple-negative and BRCA-mutated breast cancers
|Nature Structural & Molecular Biology|2010|1k
Exosome Transfer from Stromal to Breast Cancer Cells Regulates Therapy Resistance Pathways
|Cell|2014|798
Laminin receptor on platelets is the integrin VLA-6
|Nature|1988|675
Vinculin potentiates E-cadherin mechanosensing and is recruited to actin-anchored sites within adherens junctions in a myosin II–dependent manner
|The Journal of Cell Biology|2010|660